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| Bladder Drainage in Spinal Cord Injury |
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| Considerations |
by flaccid paralysis (loss of bladder tone) below the level of the injury, and loss of spinal cord reflexes including bladder and bowel sphincter control. Spinal shock often coincides with neurogenic shock, which is evidenced by loss of autonomic control (bradycardia, vasodilation, hypotension). Spinal shock can persist for several weeks, and is associated with an inability to retain urine or bowel contents. Although urinary incontinence may be present, bladder emptying is also dysfunctional, making some form of catheterization mandatory. sphincter reflex are amont the first responses to appear and are generally signs that the spinal shock phase is ending. The ability to retain urine is restored, and spastic movements of the paralyzed limbs may appear (care should be taken not to mistake these involuntary movements for a return of voluntary movement). continence of urine and feces, the cord injury prevents the individual from contracting the bladder and relaxing the sphincters to facilitate elimination. This places them at risk for autonomic dysreflexia. commence. Catheterization should be performed q 4 h initially. The goal is to perform catheterization frequency to keep bladder volume < 500 mL. Catherization frequency can be increased if adquately maintaining this goal. distention, as a full bladder can trigger autonomic dysreflexia. Bladder distention may also reduce blood supply and increase the risk for infection. Intermittent catheterization frequency should increase when fluid intake rises or diuretics are administered. incontinence. Condom drainage can be used to protect skin and linen, however, it is important to continue to perform intermittent catheterization, as complete bladder emptying will not occur. In lower cord or incomplete injuries, some patients may be trained to persist. |
O'Donnell,
W. (1987). Neurological management in patient with acute spinal cord injury.
Critical Care Clinics. July. pp
599-608.
Reed,
MA. (1987). Nursing consideration in acute spinal cord injury. Critical
Care Clinics. July. pp 680, 687-688.